Holly Van Remmen - Concepedia

Author

Holly Van Remmen

The University of Texas Health Science Center at San Antonio ORCID

Also Known As

H Van Remmen, H. Van Remmen, Heng-Keat Tam, Holly Van Remmen, Holly Van Remmen, Holly van Remmen, Holly van Remmen, Van Remmen, Holly

308

Publications

27K

Citations

H-Index

166

Concepts

All Affiliations

The University of Texas Health Science Center at San Antonio South Texas Veterans Health Care System Oklahoma Medical Research Foundation Geriatric Research Education and Clinical Center Oklahoma City VA Medical Center

About

Holly Van Remmen is an author at The University of Texas Health Science Center at San Antonio specializing in medicine, oxidative stress, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	47	81	11.2K
2	Health Sciences	19	21	2.1K
3	Natural Sciences	19	19	2K
4	Social Sciences	4	4	368
5	Engineering	1	1	26

Holly Van Remmen

169

Publications

21.5K

Citations

H-Index

Page 1

	Year	Citations
Mitochondrial DNA Mutations, Oxidative Stress, and Apoptosis in Mammalian Aging Gregory C. Kujoth, Asimina Hiona, Thomas D. Pugh, Science AgingMitochondrial FunctionMitochondrial DynamicLongevityGenetics	2005	2.1K
Extension of Murine Life Span by Overexpression of Catalase Targeted to Mitochondria Samuel E. Schriner, Nancy J. Linford, George M. Martin, Science	2005	1.7K
Complex III Releases Superoxide to Both Sides of the Inner Mitochondrial Membrane Florian L. Müller, Yuhong Liu, Holly Van Remmen Journal of Biological Chemistry	2004	1K
The selenoprotein GPX4 is essential for mouse development and protects from radiation and oxidative damage insults Levi Yant, Qitao Ran, Lin Rao, Free Radical Biology and Medicine Knockout MouseSelenoprotein Gpx4Oxidative Damage InsultsDevelopmental BiologySelenium Deficiency	2003	806
Does oxidative damage to DNA increase with age? Michelle L. Hamilton, Holly Van Remmen, Jessica A. Drake, Proceedings of the National Academy of Sciences Dna DamageGeneticsLipid PeroxidationMolecular BiologyRedox Biology	2001	759
Life-long reduction in MnSOD activity results in increased DNA damage and higher incidence of cancer but does not accelerate aging Holly Van Remmen, Yuji Ikeno, Michelle M. Hamilton, Physiological Genomics	2003	729
CuZnSOD deficiency leads to persistent and widespread oxidative damage and hepatocarcinogenesis later in life Sailaja Elchuri, Terry D. Oberley, Wenbo Qi, Oncogene BiochemistryLiver PhysiologyLipid PeroxidationWidespread Oxidative DamagePathology	2004	660
Insulin resistance is a cellular antioxidant defense mechanism Kyle L. Hoehn, Adam B. Salmon, Cordula Hohnen-Behrens, Proceedings of the National Academy of Sciences Metabolic RemodelingInsulin SignalingRedox BiologyOxidative StressMetabolic Syndrome	2009	522
Increased Oxidative Damage Is Correlated to Altered Mitochondrial Function in Heterozygous Manganese Superoxide Dismutase Knockout Mice Melissa D. Williams, Holly Van Remmen, Craig C. Conrad, Journal of Biological Chemistry	1998	452
Absence of CuZn superoxide dismutase leads to elevated oxidative stress and acceleration of age-dependent skeletal muscle atrophy Florian L. Müller, Wook Song, Yuhong Liu, Free Radical Biology and Medicine AgingBiochemistryMuscle InjurySkeletal MusclePhysiology	2006	444

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