Douglas S. Micalizzi

Author

Also Known As

D Micalizzi, D S Micalizzi, D. Micalizzi, D.S. Micalizzi, Doug S. Micalizzi, Douglas Micalizzi, Douglas S Micalizzi, Douglas S. Micalizzi, Douglas Scott Micalizzi, Micalizzi, Douglas

Publications

3.4K

Citations

H-Index

Concepts

All Affiliations

Harvard University Massachusetts General Hospital Howard Hughes Medical Institute University of Colorado Denver University of Colorado Anschutz Medical Campus

About

Douglas S. Micalizzi is an author at Harvard University specializing in medicine, tumor biology, and cancer research.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	797
2	Cell Biology	6	6	780
3	Health Sciences	2	2	227
4	Immunology	2	2	137
5	Pathology	1	1	19

Douglas S. Micalizzi

Publications

1.7K

Citations

H-Index

Page 1

	Year	Citations
Deregulation of ribosomal protein expression and translation promotes breast cancer metastasis Richard Y. Ebright, Sooncheol Lee, Ben S. Wittner, Science	2020	357
The miR-106b-25 cluster targets Smad7, activates TGF-β signaling, and induces EMT and tumor initiating cell characteristics downstream of Six1 in human breast cancer Ashley Smith, Ritsuko Iwanaga, David Drasin, Oncogene Tumor BiologyMedicineTgf-β SignalingInduces EmtCancer Cell Biology	2012	295
The Six1 homeoprotein induces human mammary carcinoma cells to undergo epithelial-mesenchymal transition and metastasis in mice through increasing TGF-β signaling Douglas S. Micalizzi, Kimberly Christensen, Paul Jedlicka, Journal of Clinical Investigation	2009	231
Eya2 is required to mediate the pro-metastatic functions of Six1 via the induction of TGF-β signaling, epithelial–mesenchymal transition, and cancer stem cell properties Susan M. Farabaugh, Douglas S. Micalizzi, Paul Jedlicka, Oncogene Tumor BiologyDevelopmental BiologyMedicineTgf-β SignalingEpithelial–mesenchymal Transition	2011	143
SIX1 induces lymphangiogenesis and metastasis via upregulation of VEGF-C in mouse models of breast cancer Chuan Wang, Paul Jedlicka, Aaron Patrick, Journal of Clinical Investigation Breast OncologyImmunologyCell ProliferationCancer BiologyTumor Biology	2012	118
Homeoprotein Six1 Increases TGF-β Type I Receptor and Converts TGF-β Signaling from Suppressive to Supportive for Tumor Growth Douglas S. Micalizzi, Chu-An Wang, Susan M. Farabaugh, Cancer Research Cell ProliferationCancer BiologyMammary Gland DevelopmentTumor BiologyTgf-β Type	2010	109
HIF1A signaling selectively supports proliferation of breast cancer in the brain Richard Y. Ebright, Marcus Zachariah, Douglas S. Micalizzi, Nature Communications	2020	102
Six1 Overexpression in Mammary Cells Induces Genomic Instability and Is Sufficient for Malignant Transformation Ricardo D. Coletta, Kimberly Christensen, Douglas S. Micalizzi, Cancer Research Breast OncologyCancer BiologyMammary Gland DevelopmentTumor BiologyIs Sufficient	2008	96
Expression of Six1 in luminal breast cancers predicts poor prognosis and promotes increases in tumor initiating cells by activation of extracellular signal-regulated kinase and transforming growth factor-beta signaling pathways Ritsuko Iwanaga, Chuan Wang, Douglas S. Micalizzi, Breast Cancer Research	2012	90
The Six1 oncoprotein downregulates p53 via concomitant regulation of RPL26 and microRNA-27a-3p Christina G. Towers, Anna L. Guarnieri, Douglas S. Micalizzi, Nature Communications	2015	57

Page 1