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Bis(propyl)-cognitin Prevents β-amyloid-induced Memory Deficits as Well as Synaptic Formation and Plasticity Impairments via the Activation of PI3-K Pathway

Plasticity ImpairmentsSynaptic PlasticityNeurodegenerative DiseasesSynaptic NeurosciencePi3-k PathwayNeuroscienceNeurologyβ-Amyloid-induced Memory DeficitsStructural PlasticityMolecular NeurobiologyMedicineSynaptic Signaling