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VEGI attenuates the inflammatory injury and disruption of blood–brain barrier partly by suppressing the TLR4/NF-κB signaling pathway in experimental traumatic brain injury

Traumatic Brain InjuryImmune RegulationImmunologyCell DeathNeuroinflammationInflammationBrain InjuryNeurologyNeuroimmunologyCell SignalingTissue InjuryMolecular SignalingInflammatory InjuryChronic InflammationBrain-immune InteractionNeuroprotectionImmune FunctionBrain Injury PreventionCell BiologyBlood–brain BarrierInflammatory SignallingMedicine