Publication | Open Access

G- to F-actin modulation by a single amino acid substitution in the actin binding site of actobindin and thymosin beta 4.

Molecular PharmacologyProtein FunctionSignal TransductionMolecular PhysiologyBiochemistryG Protein-coupled ReceptorNatural SciencesThymosin Beta 4Mechanism Of ActionMolecular BiologyReceptor (Biochemistry)Cell MotilityCytoskeletonMedicineCell BiologyCell SignalingCellular PhysiologyF-actin Modulation