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Co-exposure of arsenic and polystyrene-nanoplastics induced kidney injury by disrupting mitochondrial homeostasis and mtROS-mediated ferritinophagy and ferroptosis

Chemical EngineeringEngineeringBiochemistryIron MetabolismMitochondrial HomeostasisMtros-mediated FerritinophagyMedicineMetalloid ContaminationBioactive MetalNanotoxicologyToxicologyMetal ToxicityKidney InjuryRedox BiologyToxicological MechanismOxidative Stress