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Inhibition of p38 MAPK/NF-κB p65 signaling pathway activity by rare ginsenosides ameliorates cyclophosphamide-induced premature ovarian failure and KGN cell injury

InflammationCytokinePathway ActivityRare GinsenosidesMedicineApoptosisCell DeathP38 Mapk/nf-κb P65PharmacologyCell BiologyCell SignalingOxidative StressOvarian CancerMolecular Signaling