C-type lectin receptor expression is a hallmark of neutrophils infiltrating the skin in epidermolysis bullosa acquisita

Abstract

Collectively, our results suggest that while the upregulation of <i>Clec4n</i>, <i>Clec4d</i>, and <i>Clec4e</i> is a hallmark of activated dermal neutrophil populations, their individual contribution to the pathogenesis of EBA is dispensable.