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Irisin protects against doxorubicin-induced cardiotoxicity by improving AMPK-Nrf2 dependent mitochondrial fusion and strengthening endogenous anti-oxidant defense mechanisms

InflammationDoxorubicin-induced CardiotoxicityReductive StressOxysterolMedicineLipid PeroxidationPhysiologyCell DeathRedox BiologyReactive Oxygen SpeciePharmacologyIrisin ProtectsCell SignalingOxidative Stress