Publication | Open Access

A novel mechanism for the protection against acute lung injury by melatonin: mitochondrial quality control of lung epithelial cells is preserved through SIRT3-dependent deacetylation of SOD2

InflammationAcute Lung InjuryLung Epithelial CellsReductive StressMitochondrial Quality ControlMitochondrial FunctionLung InflammationMedicineCell DeathPulmonary PharmacologyPharmacologyCell BiologyCell SignalingLung CancerMelatoninOxidative Stress