Publication | Open Access
eNOS-derived nitric oxide regulates endothelial barrier function through VE-cadherin and Rho GTPases
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2014
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Molecular BiologyCellular PhysiologyOxidative StressInflammationAngiogenesisRho GtpasesMicrovascular DysfunctionCell SignalingAtherosclerosisCell SciHealth SciencesEndothelial Cell PathobiologyMolecular PhysiologyVascular BiologyVascular Endothelial Growth FactorEnos-derived Nitric OxideCell BiologyEndothelial Barrier FunctionSignal TransductionPhysiologyEndothelial DysfunctionTranslational ResearchCell SystemsMedicineWilliam C. Sessa
There was an error published in J. Cell Sci. 126, 5541-5552.Both Annarita Di Lorenzo and William C. Sessa should have been designated as authors for correspondence. They can be contacted at and2039@med.cornell.edu and william.sessa@yale.edu, respectively.We apologise to the readers for any confusion that this error might have caused.