Publication | Open Access
Uncompensated mitochondrial oxidative stress underlies heart failure in an iPSC-derived model of congenital heart disease
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Citations
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References
2022
Year
CardiomyopathyHeart FailureCardiovascular DiseaseBiochemistryMitochondrial FunctionGeneticsPhysiologyIpsc-derived ModelAdult Congenital Heart DiseaseRedox BiologyMedicineCardiologyCongenital Heart DiseaseOxidative Stress
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