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γ-mangostin attenuates amyloid-β42-induced neuroinflammation and oxidative stress in microglia-like BV2 cells via the mitogen-activated protein kinases signaling pathway

InflammationNeurodegenerative DiseasesMicroglia-like Bv2 CellsMedicineImmunologyBrain-immune InteractionNeuroprotectionNeuroimmunologyCell BiologyMitogen-activated ProteinOxidative StressNeuroinflammation