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Baicalin ameliorates 2,4-dinitrochlorobenzene-induced atopic dermatitis-like skin lesions in mice through modulating skin barrier function, gut microbiota and JAK/STAT pathway

InflammationImmunotoxicologyAtopic DermatitisMedicineSkin Barrier FunctionGut MicrobiotaJak/stat PathwayToxicologyDermatologyMicrobiomeExperimental DermatologyPharmacologyOxidative StressDrug Resistance