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Sepsis-induced acute lung injury in young rats is relieved by calycosin through inactivating the HMGB1/MyD88/NF-κB pathway and NLRP3 inflammasome

Acute Lung InjuryInflammatory Lung DiseaseLung InflammationImmune RegulationImmunologyPathologyInflammationDisease PathophysiologySepsisCell SignalingTissue InjuryChronic InflammationPulmonary FibrosisImmune FunctionInflammatory DiseaseHmgb1/myd88/nf-κb PathwayInflammation BiologyYoung RatsMedicine