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Kcnk3 dysfunction exaggerates the development of pulmonary hypertension induced by left ventricular pressure overload

36

Citations

31

References

2021

Year

Abstract

Our results indicate that Kcnk3-LOF is a key event in the pathobiology of PH due to AAC, suggesting that Kcnk3 channel dysfunction could play a potential key role in the development of PH due to LHD.

References

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