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Low-activity programming of the PDGFRβ/FAK pathway mediates H-type vessel dysplasia and high susceptibility to osteoporosis in female offspring rats after prenatal dexamethasone exposure

Developmental BiologyLow-activity ProgrammingPdgfrβ/fak PathwayVascular BiologyEndocrinologyMedicineCell BiologyOsteoporosisBone MetabolismOsteocalcinH-type Vessel Dysplasia