Publication | Closed Access
AI‐2 represses CagA expression and bacterial adhesion, attenuating the <i>Helicobacter pylori</i>‐induced inflammatory response of gastric epithelial cells
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Citations
60
References
2021
Year
AI-2 plays an important role in the pathogenesis of H. pylori. AI-2 inhibits the bacterial adhesion, expression, and translocation of CagA, and attenuates the inflammatory response of AGS cells induced by H. pylori.
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