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High-glucose-induced apoptosis, ROS production and pro-inflammatory response in cardiomyocytes is attenuated by metformin treatment via PP2A activation

InflammationMetabolic SyndromeMedicineApoptosisPhysiologyDiabetesCell DeathPp2a ActivationMetformin TreatmentMetabolic RegulationHigh-glucose-induced ApoptosisMetabolic RemodelingMetabolismPharmacologyCell BiologyCell SignalingOxidative StressHealth Sciences