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Excessive ROS production and enhanced autophagy contribute to myocardial injury induced by branched-chain amino acids: Roles for the AMPK-ULK1 signaling pathway and α7nAChR

Cell AutophagyCardiomyopathySignal TransductionMedicinePhysiologyAutophagyBranched-chain Amino AcidsAutophagy ContributeMetabolic SignalingExcessive Ros ProductionCell BiologyCell SignalingOxidative StressMolecular Signaling