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Elevating bioavailable iron levels in mitochondria suppresses the defective phenotypes caused by PINK1 loss-of-function in Drosophila melanogaster

BiologyDrosophila MelanogasterBioavailable Iron LevelsMitochondrial BiogenesisMitochondrial FunctionMitochondrial DynamicIron MetabolismGeneticsMitochondrial DynamicsMolecular GeneticsMetabolismMedicineRedox BiologyPink1 Loss-of-functionOxidative Stress