<scp>LncRNA HOTAIR</scp> enhances breast cancer radioresistance through facilitating <scp>HSPA1A</scp> expression via sequestering <scp>miR</scp>‐449b‐5p

Abstract

Therapeutically, HOTAIR and HSPA1A may be employed as potential targets for BRCA radiotherapy. Our findings shed new light into the mechanism by which lncRNAs modulate the radiosensitivity of tumors.