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A small nuclear acidic protein (MTI-II, Zn2+-binding protein, parathymosin) attenuates TNF-α inhibition of BMP-induced osteogenesis by enhancing accessibility of the Smad4-NF-κB p65 complex to Smad binding element

InflammationZn2+-binding ProteinSmad Binding ElementSignal TransductionMolecular PhysiologyTnf-α InhibitionImmunologyOsteogenesisBone HomeostasisMedicineCell BiologyCell SignalingBone MetabolismMolecular Signaling