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Deletion of Glycogen Synthase Kinase-3β in D2 Receptor–Positive Neurons Ameliorates Cognitive Impairment via NMDA Receptor–Dependent Synaptic Plasticity

Synaptic PlasticityNeurodegenerative DiseasesMolecular NeuroscienceSynaptic NeuroscienceSynaptic TransmissionGlycogen Synthase Kinase-3βNeuroscienceNeurologyMolecular NeurobiologyStructural PlasticityMedicineSynaptic SignalingSocial Sciences