Publication | Closed Access

Granulocytic myeloid-derived suppressor cells contribute to IFN-I signaling activation of B cells and disease progression through the lncRNA NEAT1-BAFF axis in systemic lupus erythematosus

InflammationAutoimmune DiseaseSystemic Lupus ErythematosusImmune Cell DevelopmentImmunologyB CellsImmunologic MechanismAutoimmunityInnate ImmunityImmunopathologyImmunotherapyMedicineCell BiologyCell SignalingLncrna Neat1-baff Axis