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Lipopolysaccharide exposure in a rat sepsis model results in hippocampal amyloid-β plaque and phosphorylated tau deposition and corresponding behavioral deficits

InflammationDisease PathophysiologyLipopolysaccharide ExposureTau DepositionBrain-immune InteractionBrain InjuryNeurologyNeuroscienceMedicineHippocampal Amyloid-β PlaqueNeuroinflammation