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Methyl jasmonate abrogates rotenone-induced parkinsonian-like symptoms through inhibition of oxidative stress, release of pro-inflammatory cytokines, and down-regulation of immnopositive cells of NF-κB and α-synuclein expressions in mice

InflammationNeurodegenerative DiseasesImmnopositive CellsNeurobiology Of DiseaseAnti-inflammatoryMedicineImmunologyDegenerative PathologyNeuroprotectionNeurologyNeurodegenerationMethyl JasmonatePharmacologyOxidative StressNeuroinflammation