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A C-terminal peptide from secreted amyloid precursor protein-α enhances long-term potentiation in rats and a transgenic mouse model of Alzheimer's disease

Neurodegenerative DiseasesSynaptic PlasticityAlzheimer's DiseaseMolecular NeuroscienceSynaptic TransmissionDegenerative PathologyProtein MisfoldingNeuroscienceTransgenic Mouse ModelMedicineSynaptic SignalingC-terminal PeptideSocial SciencesBiomolecular Engineering