Baicalin suppresses proliferation, migration, and invasion in human glioblastoma cells via Ca<sup>2+</sup>-dependent pathway

Abstract

Our observations suggest that baicalin exerts cytotoxic effects on human glioblastoma cells by the autophagy-related apoptosis through Ca²⁺ movement to the cytosol. Furthermore, baicalin has the potential as a candidate for the treatment of glioblastoma.