The association of <i>HBG2</i>,<i> BCL11A,</i> and <i>HMIP</i> polymorphisms with fetal hemoglobin and clinical phenotype in Iraqi Kurds with sickle cell disease

Abstract

It was demonstrated that SNPs in all three major HbF QTLs contribute significantly to HbF and clinical variability in Iraqi Kurds with SCD and that the cumulative number of minor alleles at contributing SNPs may serve as a better predictor of such variability in this population.