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Chlamydia pneumoniae infection promotes monocyte transendothelial migration by increasing vascular endothelial cell permeability via the tyrosine phosphorylation of VE-cadherin

InflammationEndothelial Cell PathobiologyTyrosine PhosphorylationMonocyte Transendothelial MigrationPathogenesisImmunologyChlamydia Pneumoniae InfectionEndothelial DysfunctionVascular BiologyMedicineCell BiologyPhagocyte