Publication | Open Access
<i>De novo</i> variants in <i><scp>CAMK</scp>2A</i> and <i><scp>CAMK</scp>2B</i> cause neurodevelopmental disorders
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Citations
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References
2018
Year
Our data highlight the importance of CaMKII <i>α</i> and CaMKII <i>β</i> and their autoinhibitory regulation in human brain function, and suggest the enhancement of A-type K<sup>+</sup> currents as a possible pathophysiological basis.
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