Publication | Open Access

p66Shc deficiency enhances CXCR4 and CCR7 recycling in CLL B cells by facilitating their dephosphorylation-dependent release from β-arrestin at early endosomes

Signal TransductionEarly EndosomesSignaling PathwayCell RegulationCell SignalingImmunologyEndocytic PathwayCll B CellsCellular BiochemistryMedicineCell BiologyCellular PhysiologyDephosphorylation-dependent Release