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FSP1-specific SMAD2 knockout in renal tubular, endothelial, and interstitial cells reduces fibrosis and epithelial-to-mesenchymal transition in murine STZ-induced diabetic nephropathy

Fsp1-specific Smad2 KnockoutRenal PathologyRenal InflammationInflammationInterstitial CellsKidney Tubule RemodelingChronic Kidney DiseaseCell SignalingRenal TubularFibrosisMolecular PhysiologyVascular BiologyRenal PathophysiologyCell BiologyUrologyDiabetesDiabetic Kidney DiseaseMedicineNephrologyKidney Research