Publication | Open Access

Inflammatory mediators mediate airway smooth muscle contraction through a G protein-coupled receptor–transmembrane protein 16A–voltage-dependent Ca2+ channel axis and contribute to bronchial hyperresponsiveness in asthma

AsthmaInflammationSignal TransductionInflammatory Lung DiseaseMolecular PhysiologyLung InflammationG Protein-coupled ReceptorPhysiologyImmunologySmooth Muscle ContractionReceptor (Biochemistry)Ion ChannelsPulmonary PhysiologyPulmonary PharmacologyInflammatory MediatorsMedicineCell SignalingBronchial Hyperresponsiveness