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Chronic exposure to tumor necrosis factor in vivo induces hyperalgesia, upregulates sodium channel gene expression and alters the cellular electrophysiology of dorsal root ganglion neurons

InflammationSpinal Cord InjuryHealth SciencesNeurophysiologyPain MedicineMedicinePhysiologyCellular ElectrophysiologyNecrosis FactorNeuropathic PainCell DeathMolecular PainPain MechanismPeripheral NerveChronic ExposureNervous SystemPharmacologyPain Research