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Endothelial deletion of mTORC1 protects against hindlimb ischemia in diabetic mice via activation of autophagy, attenuation of oxidative stress and alleviation of inflammation

InflammationHindlimb IschemiaMitophagyCell AutophagyAtherosclerosisDiabetesAutophagyPhysiologyImmunologyEndothelial DysfunctionVascular BiologyMicrovascular DysfunctionMedicineCell BiologyCell SignalingMtorc1 ProtectsOxidative Stress