Concepedia

Publication | Closed Access

CFTR‐dependent chloride efflux in cystic fibrosis mononuclear cells is increased by ivacaftor therapy

22

Citations

40

References

2017

Year

Abstract

In patients with non-G551D mutations, ivacaftor improved both chloride transport in sweat ducts and chloride efflux in MNC, that is, functions directly imputed to CFTR.

References

YearCitations

Page 1