Publication | Open Access

Reversal of high fat diet-induced obesity improves glucose tolerance, inflammatory response, β-amyloid accumulation and cognitive decline in the APP/PSEN1 mouse model of Alzheimer's disease

ObesityMetabolic SyndromeNeurodegenerative DiseasesAlzheimer's DiseaseApp/psen1 Mouse ModelMedicinePhysiologyInflammatory ResponseVascular DementiaNeurologyNeuroscienceAging-associated DiseaseGlucose ToleranceHealth Sciences