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Dioxin-like rather than non-dioxin-like PCBs promote the development of endometriosis through stimulation of endocrine–inflammation interactions

InflammationUterine FibroidsNon-dioxin-like PcbsEndocrine MechanismMedicineUterine ReceptivityImplantation (Embryology)GynecologyEndocrine–inflammation InteractionsMenstrual CycleEndocrinologyPharmacologyWomen's Health