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Neuronal Deletion of Kmt2a/Mll1 Histone Methyltransferase in Ventral Striatum is Associated with Defective Spike-Timing-Dependent Striatal Synaptic Plasticity, Altered Response to Dopaminergic Drugs, and Increased Anxiety

Synaptic PlasticityMolecular NeurosciencePsychiatryNeurotransmitterKmt2a/mll1 Histone MethyltransferaseNeuropharmacologyNeurologyNeuroscienceVentral StriatumDopaminergic DrugsDopamineMedicineNeurochemistryMolecular NeurobiologySocial Sciences