Umbilical venous erythropoietin and umbilical arterial pH in relation to morphologic placental abnormalities

Abstract

Fetal erythropoietin production is stimulated by hypoxia after a few hours' delay and leads to increased erythropoiesis. Placental examination combined with measurement of umbilical venous erythropoietin and umbilical arterial pH provides information about earlier fetal hypoxia.