<i>Porphyromonas gingivalis</i>‐induced production of reactive oxygen species, tumor necrosis factor‐α, interleukin‐6, <scp>CXCL</scp>8 and <scp>CCL</scp>2 by neutrophils from localized aggressive periodontitis and healthy donors: modulating actions of red blood cells and resolvin E1

Abstract

Our data support that binding to RBCs protects P. gingivalis from ROS and concomitantly enhances neutrophil release of proinflammatory cytokines providing a selective advantage for P. gingivalis growth.