mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis - Concepedia

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mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis

51

Citations

57

References

2016

Year

Laetitia Mazelin, Baptiste Panthu, Anne-Sophie Nicot, Edwige Belotti, Lionel Tintignac, Geoffrey Teixeira, Qing Zhang, Valérie Risson, Dominique Baas, Emilie Delaune,

Journal of Molecular and Cellular Cardiology

Translation DefectsCardiac MuscleCardiomyopathyHeart FailureMolecular PhysiologyInfant MiceCell SignalingPhysiologyApoptosisCell DeathCardiologyMtor InactivationMedicineCell BiologyCellular PhysiologyCardiovascular Translational Research

References

	Year	Citations

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