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Transforming growth-beta 1 contributes to isoflurane postconditioning against cerebral ischemia–reperfusion injury by regulating the c-Jun N-terminal kinase signaling pathway

Growth-beta 1Cerebral Ischemia–reperfusion InjuryOxidative StressNeuroinflammationInflammationCerebral Vascular RegulationBrain InjuryNeurologyC-jun N-terminal KinaseCell SignalingIschemic SyndromeHealth SciencesMolecular SignalingVascular BiologyNeuroprotectionCerebral Blood FlowReperfusion InjuryCell BiologyPhysiologyMedicine