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The misfolded pro-inflammatory protein S100A9 disrupts memory via neurochemical remodelling instigating an Alzheimer’s disease-like cognitive deficit

Neurodegenerative DiseasesDisease-like Cognitive DeficitAlzheimer's DiseaseMedicineImmunologyDegenerative PathologyNeurochemical BiomarkersAlzheimer ’Brain-immune InteractionNeuroscienceNeurologyNeurodegenerationNeuroimmunologySynaptic SignalingProtective MechanismsNeuroinflammation