Publication | Open Access

Soluble prion protein and its N-terminal fragment prevent impairment of synaptic plasticity by Aβ oligomers: Implications for novel therapeutic strategy in Alzheimer's disease

Neurodegenerative DiseasesSynaptic PlasticityAlzheimer's DiseaseSynaptic NeuroscienceSynaptic TransmissionMolecular BiologyDegenerative PathologyPrion DiseaseSoluble Prion ProteinProtein MisfoldingNeurologyNeuroscienceSynaptic DysfunctionAβ OligomersMolecular NeurobiologyMedicineSynaptic Signaling