Publication | Open Access

Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis

MechanobiologyCardiac MuscleCardiomyopathyHeart FailureCardiac ReprogrammingHypertrophic ResponseCardiovascular DiseasePressure-overloaded HeartPhysiologyCardiac FibrosisCardiovascular FunctionMedicineCell BiologyCardiologyCardiac MechanicCardiac Pathology