Publication | Open Access

Anti-glycan antibodies halt axon regeneration in a model of Guillain Barrè Syndrome axonal neuropathy by inducing microtubule disorganization via RhoA–ROCK-dependent inactivation of CRMP-2

NeuroregenerationNeurobiology Of DiseaseMolecular NeurosciencePeripheral NervesMedicineExperimental NeuropathologyImmunologyNeuromodulation TherapiesAxon RegenerationAnti-glycan AntibodiesNeurologyNeuroscienceNeuropathologyNeuroimmunologySynaptic SignalingSocial SciencesRhoa–rock-dependent Inactivation