Cross‐reactivity of GroEL antibodies with human heat shock protein 60 and quantification of pathogens in atherosclerosis

Abstract

These results support the hypothesis that in some patients, cross-reactivity of the immune response to bacterial HSPs including those of periodontal pathogens, with arterial endothelial cells expressing hHSP60 may be a possible mechanism for the association between atherosclerosis and periodontal infection.